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KMT2D haploinsufficiency in Kabuki Syndrome disrupts neur... | ResearchHub
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KMT2D haploinsufficiency in Kabuki Syndrome disrupts neuronal function through transcriptional and chromatin rewiring independent of H3K4-monomethylation
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Authors
Michele Gabriele
25 more
Michele Gabriele
•
Alessandro Vitriolo
23 more
•
Giuseppe Testa
Published
April 23, 2021
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Preprint Server
Topics
Biology
Neuroscience
Genetics And Genomics
Genetics
Gene
Show all topics
DOI
10.1101/2021.04.22.440945
License
CC-BY-NC-ND
Other Formats
PDF
Supporters
Support the authors with ResearchCoin
Tip RSC
Preprint Server
Topics
Biology
Neuroscience
Genetics And Genomics
Genetics
Gene
Show all topics
DOI
10.1101/2021.04.22.440945
License
CC-BY-NC-ND
Other Formats
PDF
