Deficits in mitochondrial function and glucose metabolism seen in sporadic and familial Alzheimers disease derived Astrocytes are ameliorated by increasing hexokinase 1 expression.

Authors

Simon Bell

Published

March 25, 2023

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10.1101/2023.03.23.534020

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Deficits in mitochondrial function and glucose metabolism seen in sporadic and familial Alzheimers disease derived Astrocytes are ameliorated by increasing hexokinase 1 expression.

Authors

Simon Bell

Published

March 25, 2023

Add a comment...

Supporters

Support the authors with ResearchCoin

Preprint Server

Topics

DOI

10.1101/2023.03.23.534020

License

CC-BY-ND

Other Formats

PDF

Supporters

Support the authors with ResearchCoin

Preprint Server

Topics

DOI

10.1101/2023.03.23.534020

License

CC-BY-ND

Other Formats

PDF

Paper

Paper

Paper

Paper

Deficits in mitochondrial function and glucose metabolism seen in sporadic and familial Alzheimers disease derived Astrocytes are ameliorated by increasing hexokinase 1 expression.

Supporters

Preprint Server

Topics

DOI

License

Other Formats

Deficits in mitochondrial function and glucose metabolism seen in sporadic and familial Alzheimers disease derived Astrocytes are ameliorated by increasing hexokinase 1 expression.

Supporters

Preprint Server

Topics

DOI

License

Other Formats

Supporters

Preprint Server

Topics

DOI

License

Other Formats