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HTLV-1 Rex hijacks UPF1 in a CRM1 dependent manner, leadi... | ResearchHub
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HTLV-1 Rex hijacks UPF1 in a CRM1 dependent manner, leading to NMD inhibition and revealing unexpected proviral roles of UPF1
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Authors
Léa Prochasson
22 more
Léa Prochasson
•
Makram Mghezzi-Habellah
20 more
•
Vincent Mocquet
Published
June 20, 2023
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Preprint Server
Topics
Biology
Immunology
Paleontology
Molecular Biology
Genetics
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DOI
10.1101/2023.06.20.545693
License
cc-no
Supporters
Support the authors with ResearchCoin
Tip RSC
Preprint Server
Topics
Biology
Immunology
Paleontology
Molecular Biology
Genetics
Show all topics
DOI
10.1101/2023.06.20.545693
License
cc-no