Mitochondria-targeted hydrogen sulfide donor reduces fatty liver and obesity in mice fed a high fat diet by inhibiting de novo lipogenesis and inflammation via mTOR/SREBP-1 and NF-κB signaling pathways

Authors

Aneta Stachowicz

Published

April 17, 2024

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10.1101/2024.04.17.589169

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Mitochondria-targeted hydrogen sulfide donor reduces fatty liver and obesity in mice fed a high fat diet by inhibiting de novo lipogenesis and inflammation via mTOR/SREBP-1 and NF-κB signaling pathways

Authors

Aneta Stachowicz

Published

April 17, 2024

Add a comment...

Supporters

Support the authors with ResearchCoin

Preprint Server

Topics

DOI

10.1101/2024.04.17.589169

License

cc-no

Supporters

Support the authors with ResearchCoin

Preprint Server

Topics

DOI

10.1101/2024.04.17.589169

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cc-no

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Paper

Paper

Paper

Mitochondria-targeted hydrogen sulfide donor reduces fatty liver and obesity in mice fed a high fat diet by inhibiting de novo lipogenesis and inflammation via mTOR/SREBP-1 and NF-κB signaling pathways

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Preprint Server

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DOI

License

Mitochondria-targeted hydrogen sulfide donor reduces fatty liver and obesity in mice fed a high fat diet by inhibiting de novo lipogenesis and inflammation via mTOR/SREBP-1 and NF-κB signaling pathways

Supporters

Preprint Server

Topics

DOI

License

Supporters

Preprint Server

Topics

DOI

License