In the absence of endogenous IL-10, mice acutely infected with Toxoplasma gondii succumb to a lethal immune response dependent on CD4+ T cells and accompanied by overproduction of IL-12, IFN-gamma and TNF-alpha.

Authors

Ricardo Gazzinelli

Published

July 15, 1996

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Journal

The Journal of Immunology

Topics

DOI

10.4049/jimmunol.157.2.798

In the absence of endogenous IL-10, mice acutely infected with Toxoplasma gondii succumb to a lethal immune response dependent on CD4+ T cells and accompanied by overproduction of IL-12, IFN-gamma and TNF-alpha.

Authors

Ricardo Gazzinelli

Published

July 15, 1996

Add a comment...

Supporters

Support the authors with ResearchCoin

Journal

The Journal of Immunology

Topics

DOI

10.4049/jimmunol.157.2.798

Paper

Paper

Paper

Paper

In the absence of endogenous IL-10, mice acutely infected with Toxoplasma gondii succumb to a lethal immune response dependent on CD4+ T cells and accompanied by overproduction of IL-12, IFN-gamma and TNF-alpha.

Supporters

Journal

Topics

DOI

In the absence of endogenous IL-10, mice acutely infected with Toxoplasma gondii succumb to a lethal immune response dependent on CD4+ T cells and accompanied by overproduction of IL-12, IFN-gamma and TNF-alpha.

Supporters

Journal

Topics

DOI