NLRP3- and AIM2-autonomy in a mouse model of MSU crystal-induced acute inflammation in vivo suggests imiquimod-dependent targeting of Il-1 expression as relevant therapy for gout patients.

Authors

Alexandre Mariotte

Published

January 28, 2020

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10.1101/772756

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NLRP3- and AIM2-autonomy in a mouse model of MSU crystal-induced acute inflammation in vivo suggests imiquimod-dependent targeting of Il-1 expression as relevant therapy for gout patients.

Authors

Alexandre Mariotte

Published

January 28, 2020

Add a comment...

Supporters

Support the authors with ResearchCoin

Preprint Server

Topics

DOI

10.1101/772756

License

cc-no

Supporters

Support the authors with ResearchCoin

Preprint Server

Topics

DOI

10.1101/772756

License

cc-no

Paper

Paper

Paper

Paper

NLRP3- and AIM2-autonomy in a mouse model of MSU crystal-induced acute inflammation in vivo suggests imiquimod-dependent targeting of Il-1 expression as relevant therapy for gout patients.

Supporters

Preprint Server

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DOI

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NLRP3- and AIM2-autonomy in a mouse model of MSU crystal-induced acute inflammation in vivo suggests imiquimod-dependent targeting of Il-1 expression as relevant therapy for gout patients.

Supporters

Preprint Server

Topics

DOI

License

Supporters

Preprint Server

Topics

DOI

License